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May 2023
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Asfixia perinatal fisiopatologia pdf1/20/2023 We conclude that endogenous opioids inhibit the vasoconstriction of these vascular beds during fetal asphyxia. In contrast, kidney and carcass blood flows decreased, and their vascular resistances increased. Heart, brain, and adrenal blood flows increased due to the increase in blood pressure, with no changes in their vascular resistances. Asphyxia caused hypertension, and the fetal arterial blood pressure further increased when asphyxiated fetuses received naloxone. Naloxone solution or the solvent alone was added for the last 20 min. Fetal asphyxia was induced by reducing the uterine blood flow with an inflatable occluder around the common internal iliac artery to approximately 50% of control for 40 min. La presente revisión tiene el fin de recordar la etiopatogenia, fisiopatología, expresión clínica y. Su principal expresión clínica usada como sinónimo es la encefalopatía hipóxico isquemica. Thirteen fetal sheep (0.8-0.9 of gestation) were chronically catheterized. La asfixia neonatal sigue siendo un problema importante de mortalidad y morbilidad a largo plazo en las unidades de neonatología, a pesar de las mejoras en estudio y monitoreo perinatal. We examined the hypothesis that endogenous opioids modify the cardiac output distribution during asphyxia due to changes in the vascular resistance of some fetal organs. Intravenous administration of the opioid receptor antagonist naloxone to asphyxiated fetal sheep increases the arterial blood pressure.
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